| Annotation |
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| Description | jgi|Araly1|359111|fgenesh1_pg.C_scaffold_9000151 |
| Sequence |
ATGGCCTATA ACAATTTCAC CCGCCTCGAT GCTCAATCCG CCGCCAAGAA AGCCGTCTCA GTGATCGGAT TAGGTTACTA TCTCTGCTCC GATGTTCGTT TCTCCGCTTG TAAAACTACA CCAGACGGTT CCAGGTTAGT CGAAATCGTC CCGACCCGGA ACCGTGACTT AGTCTTCCCC GGTGGGATCG TCGTTAACAA CGTCTCGAGC TCGATTAAGT GCGATAAAGG CAAACGTACT CGATTACGCT CCGATATACT CTCTTTTAAT CAGATGTCGG AGAAGTTTAA TCAAGATATG TGTCTCTCCG GGAAGATTCC GTCGGGGATG TTTAATAATA TGTTTGCGTT TAGTAAATGT TGGCCTAAAG ATGCGAGCTC TGTGAAGAAT TTAGCTTATT GGTTCATTAG CTTGTATATA CGTGTTGAGA TTGTTAGGAA GCAATTGACT CTACGTGATG AAGTGAAACG TGAGGTTCCT TCTTCTTGGT ACTCATGTAG TTGTTGGAGT GAGTACTAA |
| Length | 519 |
| Protein Sequence | |
| Protein Residue | |
| Map Location | scaffold_1: 3023625.. 3024220 |
| Download Sequence |
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| A.thaliana |
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| AGI-code | AT1G28380.1 |
| Description | MAC/Perforin domain-containing protein |
| TAIR curator summary | This gene is predicted to encode a protein involved in negatively regulating salicylic acid-related defense responses and cell death programs. nsl1 mutants develop necrotic lesions spontaneously and show other features of a defense response, such as higher levels of SA and disease resistance-related transcripts, in the absence of a biotic stimulus. The NSL1 protein is predicted to have a MACPF domain, found in proteins that form a transmembrane pore in mammalian immune responses. NSL1 transcript levels do not appear to change in response to biotic stresses, but are elevated by cycloheximide in seedlings, and by sodium chloride in roots. |
| Alias | [NSL1][necrotic spotted lesions 1] |
| Gene coexpression |
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| MR |
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| RNA-seq |
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Seasonal expression (July 2011 to June 2013) |
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| The four seasons |
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| Growth Chamber expression |
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